The Connection Between Sperm And Obesity

The Connection Between Sperm And Obesity

The Connection Between Sperm And Obesity

One issue to consider if you are experiencing male infertility is obesity. Studies have shown that male obesity has almost tripled throughout the past 30 years. It has given rise to a proportional amount of men having problems with fertility. What is the reason?

In the past five years research has proven that it is associated with a verifiable change in the oocyte that in turn can adversely affect the embryo’s development. It also can reduce the ability to establish pregnancy within in vitro fertilization efforts. More recently, the impact of obesity in male partners has been studied. Couples with an obese male have had a more difficult time getting pregnant than couples with two non-obese partners.

Evidence is emerging that proves there is a definite connection between low sperm count and being overweight. Obesity can influence sperm quality and also change its actual molecular structure. Due to how quickly the problem of obesity is growing, this is making it a primary concern of medical professionals everywhere. Studies are suggesting that obesity in males changes the health of the embryo by altering the molecular composition of sperm.

It is commonly accepted that during gestation, nutritional challenges affect the fetus, including increasing their susceptibility to future chronic diseases. Studies have proven that obese fathers have a greater chance of having obese children also. While sperm parameters including motility and concentration are critical to assessing male fertility, so is the molecular structure and contents of sperm cells. These are both needed to form healthy cells that can generate full pregnancy.

There are different models to measure the efficacy of sperm—TUNEL, SCSA, COMET, etc.—but what each shows is that there is a connection between obesity and a reduction in sperm integrity. Part of the issue is that additional unhealthy weight in a father changes the oxidative stress of sperm. Sperm are produced from the time of puberty until death. They are created as led by sex steroids, which are regulated by the pituitary gland, hypothalamus and cells within the testes.

Semen analysis studies are proving that BMI (body mass index) is associated with a reduction in plasma concentration that increases testosterone and estrogen. The tie between weight loss and sperm count is closely related. That increase of estrogen has been associated with continued infertility. While it is a proven factor of influence, there is a level of reversibility to the connection. Simple interventions like increased exercise or change of diet can reverse obesity and also the effects of it to the sperm’s viability. It is emerging still in science how much of an impact obesity has on sperm.

Data thus far however is proving that there is a connection of the father’s nutritional status and its resulting offspring’s developmental growth. With a change in diet and exercise, there is some evidence that reversing issues is a definite possibility.

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